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#nafld
disabledprincesses · 6 months
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Visual ID: one man, edited so that there’s two of them, sits in a bedroom facing, presumably, a computer. The man behind the one closest to the computer holds a fake gun, pointing it at the first man. The first man has text over him that reads “Younger me hoping I could have a cool disability like a robot replacement leg,” while the second man with the gun has text that reads “Adult me with POTS, autism, seizures, CFS/ME, FND, psychotic depression, NAFLD, etc.” end ID.
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yourblogpostwriter · 22 days
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Food for removing NAFLD.
NAFLD = Non alcoholic fatty liver disease.
This is my diet chart for NAFLD.
This is not perfectly ideal. But I can manage these foods in my poor economic conditions.
1) smashed papaya.
2) cucumber-tomato-naga 🥗 salad.
3) walnut [akh root].
4) 2 milk thistle supplement. . . .
I wish, I can add extra-vergin olive oil.
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goth-maudra · 5 months
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The first pic is on the post of an earring. Small. And I FUCKING HATE IT. It's a spore of Aspergillus fungus. We found out it's in our house. It's made me very sick. And it took our beautiful asshole away from us.
Midnight had a human-level intelligence and was a delightful jerk. She was 15. Aspergillus fungus and orbital/sinus cancer. The vet assured us that we did the kind thing today.
And now I've got leverage, in a way. I've been having Aspergilliosis symptoms for months now. Can't shake it because I have multiple immune disorders. Even had an Infectious Diseases specialist tell me I had Delusional Parasitosis.
My dead cat would beg to differ, bitch.
Her saying that put me in a deep hellscape for 3 days, because I thought I didn't know what was real. Then I thought "hey, what the actual fuck," and started advocating better...remembering all the SCORES of ways the US health system has failed me.
I plan on being a SEVERE asshole in plenty of asses.
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orcadigitals · 7 months
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Nafld Diet Pdf With The Latest Nafld Dietary Guidelines And Recipes Reco...
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squishmelo · 1 year
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MORE potions for sober people!! MORE POTIONS, MORE CONCOCTIONS!!!!!
I want to drink “potion of time stop”, I want to drink thing that looks like a bog, I want something that’s not just juice plus soda!!!
SOBER PEOPLE DESERVE CONCOCTION TOO!!!!
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mcatmemoranda · 2 years
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Definitions – Nonalcoholic fatty liver disease (NAFLD) refers to the presence of hepatic steatosis when no other causes for secondary hepatic fat accumulation (eg, heavy alcohol consumption (figure 1)) are present. NAFLD may progress to cirrhosis and is likely an important cause of cryptogenic cirrhosis. (See 'Definitions' above.)
NAFLD is subdivided into nonalcoholic fatty liver (NAFL) and nonalcoholic steatohepatitis (NASH). In NAFL, hepatic steatosis is present without evidence of significant inflammation, whereas in NASH, hepatic steatosis is associated with hepatic inflammation that may be histologically indistinguishable from alcohol-associated steatohepatitis.
●Clinical manifestations – Most patients with NAFLD are asymptomatic, although some patients with NASH may complain of fatigue, malaise, and vague right upper abdominal discomfort. Patients are more likely to come to attention because laboratory testing revealed elevated liver aminotransferases or hepatic steatosis was detected incidentally on abdominal imaging. (See 'Clinical manifestations' above.)
Patients with NAFLD may have mild or moderate elevations in the aspartate aminotransferase and alanine aminotransferase, although normal aminotransferase levels do not exclude NAFLD. (See 'Laboratory findings' above.)
Radiographic findings in patients with NAFLD include increased echogenicity on ultrasound, decreased hepatic attenuation on computed tomography, or an increased fat signal on magnetic resonance imaging. (See 'Radiographic findings' above.)
●Diagnosis – A definitive diagnosis of NAFLD requires all of the following (see 'Diagnosis' above):
•Demonstration of hepatic steatosis by imaging or biopsy
•Exclusion of significant alcohol consumption
•Exclusion of other causes of hepatic steatosis
•Absence of coexisting chronic liver disease
Radiologic findings are often sufficient to make a diagnosis of NAFLD, provided other causes of hepatic steatosis have been excluded. However, liver biopsy may be indicated if the diagnosis is not clear or to assess the degree of hepatic injury. (See 'Radiographic examinations' above and 'Role of liver biopsy' above.)
●Differential diagnosis – Other causes of hepatic steatosis include (see 'Differential diagnosis' above):
•Significant alcohol use
•Hepatitis C (particularly genotype 3)
•Wilson disease
•Lipodystrophy
•Starvation
•Parenteral nutrition
•Abetalipoproteinemia
•Medications
•Reye syndrome
•Acute fatty liver of pregnancy
•HELLP (hemolytic anemia, elevated liver enzymes, low platelet count) syndrome
•Inborn errors of metabolism
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The rising predominance of stoutness has made non-alcoholic (NAFLD) the most widely recognized ongoing liver sickness. As an outcome, NAFLD and particularly its provocative structure non-alcoholic steatohepatitis (NASH)
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squishysunflower · 1 month
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When i was told i had NASH (liver disease), i thought that my life as i knew it was over. I was so wrong it just went in a new direction. I still haven't met anyone with NASH (or NAFLD), but that doesn't change anything. I am me, and that's what matters.
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svasthvida · 3 months
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Incredible care during my Fatty Liver. Gratitude to the team for their expertise and compassion. Effective treatment, prompt recovery, and unwavering support. Highly recommend it for anyone facing health challenges. 𝐂𝐨𝐧𝐭𝐚𝐜𝐭 𝐔𝐬 𝐭𝐨 𝐁𝐞𝐠𝐢𝐧 𝐘𝐨𝐮𝐫 𝐀𝐲𝐮𝐫𝐯𝐞𝐝𝐢𝐜 𝐇𝐞𝐚𝐥𝐢𝐧𝐠! ➡️𝐂𝐚𝐥𝐥/𝐖𝐡𝐚𝐭𝐬𝐀𝐩𝐩: 099143 21828 ➡️𝐄-𝐦𝐚𝐢𝐥: [email protected]
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Attend the CME/CPD accredited accredited 14th World Gastroenterology, IBD & Hepatology Conference from December 17-19, 2024, in Dubai, UAE & Virtual, Hurry up register now. WhatsApp us: https://wa.me/442033222718?text= Register here: https://gastroenterology.universeconferences.com/registration/
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preponias · 7 months
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Non-Alcoholic Fatty Liver Disease (NAFLD)
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nammythings · 7 months
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Navigating Non-Alcoholic Fatty Liver and Enlarged Liver
In recent years, the prevalence of non-alcoholic fatty liver disease (NAFLD) and enlarged liver has seen a concerning rise, particularly among women. Once considered a condition primarily associated with excessive alcohol consumption, NAFLD is now striking individuals who abstain from alcohol. Rather than a disease, an enlarged liver is a sign of an underlying problem, such as liver disease,…
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orcadigitals · 7 months
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Fruits Are Bad For The Liver - Latest Research Reveals Which Fruit Is No...
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otaviogilbert · 8 months
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Discover the Shocking Truth about Fatty Liver
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Fatty liver, a condition characterized by the accumulation of fat in liver cells, can often go unnoticed but has significant health implications. It's essential to understand that fatty liver can progress to more severe conditions, such as non-alcoholic fatty liver disease (NAFLD) and even non-alcoholic steatohepatitis (NASH), which can lead to liver inflammation and damage. Factors like poor diet, sedentary lifestyle, obesity, and certain medical conditions contribute to its development. Educating yourself about the risks and potential consequences of fatty liver is crucial. If you suspect you might have fatty liver or related conditions, seeking medical advice is important for proper diagnosis and guidance on managing your liver health.
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artisticdivasworld · 8 months
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NAFLD and Nursing: Essential Information and Interventions
What is Non-Alcoholic Fatty Liver Disease (NAFLD)? NAFLD stands for Non-Alcoholic Fatty Liver Disease and is one of the most common liver disorders globally. The liver plays an essential role in many body functions, from producing bile that helps digest fats to storing glucose for energy. NAFLD occurs when there is an accumulation of excess fat in liver cells, that is not due to significant…
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mcatmemoranda · 4 months
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General measures – The following general measures apply to patients with nonalcoholic fatty liver disease (NAFLD):
•For patients without serologic evidence of immunity, vaccination for hepatitis A virus and hepatitis B virus.
•Standard, age-appropriate immunizations.
•For patients with hyperlipidemia, lipid-lowering therapy.
•For patients with diabetes, optimizing blood glucose control.
●Weight management – We recommend weight loss for patients with obesity (Grade 1B). Weight loss has been associated with histologic improvement in patients with NAFLD. We advise patients to lose a minimum of five to seven percent of body weight at a rate of 0.5 to 1.0 kg per week (1 to 2 lb per week) through lifestyle modifications, including dietary therapy and exercise.
●Avoiding alcohol – For patients with NAFLD, we recommend refraining from heavy alcohol consumption (Grade 1A) and suggest abstinence from alcohol (Grade 2C). Heavy alcohol use is associated with alcohol-related liver disease and other adverse consequences, including cancers of the mouth and esophagus. In patients with or at risk for NAFLD, heavy alcohol use is associated with hepatic steatosis, hepatic injury, and fibrosis progression. Whether light to moderate alcohol consumption is harmful remains somewhat uncertain as data are mixed. In the absence of definitive data, we suggest abstinence from alcohol for patients with NAFLD.
●Medical therapy
•Patients with NASH but without diabetes mellitus – For patients with biopsy-proven nonalcoholic steatohepatitis (NASH) and fibrosis stage ≥2 but without diabetes, we suggest using vitamin E (800 international units per day) (Grade 2C). Limited evidence supports a benefit of vitamin E in patients without diabetes, but some observational studies suggest a possible increase in all-cause mortality with higher dose vitamin E. As a result, we discuss the risks and benefits with the patient before starting treatment.
•Patients with NASH and diabetes mellitus – For patients with NASH and diabetes mellitus, the presence of NASH can inform the choice of glucose-lowering therapy. Although initial therapy for type 2 diabetes mellitus is typically with metformin, the beneficial impact on liver histology with certain other insulin-sensitizing agents could be a consideration when choosing a second-line agent for patients with NASH who cannot take metformin or need additional glucose-lowering therapy. In this setting, pioglitazone and GLP-1 receptor agonists (eg, liraglutide, semaglutide) are reasonable options.
●Laboratory monitoring – We obtain serum aminotransferases (alanine aminotransferase and aspartate aminotransferase) three and six months after patients with NAFLD implement lifestyle interventions for weight loss. If the aminotransferases do not return to normal levels with weight loss or if they increase, we evaluate for an alternative cause of liver disease.
●Monitoring for fibrosis – For patients with biopsy-proven NASH, we obtain a noninvasive assessment for advanced fibrosis at a time interval determined by their clinical course:
•For patients who have not been able to lose at least five to seven percent of their body weight and/or have elevated serum aminotransferases, we obtain a noninvasive assessment every three years.
•For patients who achieve their weight loss goals and have normal serum aminotransferases, we obtain a noninvasive assessment every four years.
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