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dr-smita-goel-homeopathy-clinic · 1 month

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A person with Raynaud's disease experiences pain in the extremities, for example, the fingers, when temperatures drop.

Blood vessels narrow and almost completely shut down. Fingers or toes turn from white to blue and, then, as the blood returns, they flush red.

Also known as Raynaud's syndrome or Raynaud's phenomenon, As per research females are an estimated nine times more likely to be affected than males.

Fast facts on Raynaud's disease are followings:

• Raynaud's disease is caused by peripheral blood vessels overreacting to cold.

• Maurice Raynaud first described the disease in 1862.

• Females and people living in colder climates are more often affected.

• A capillaroscopy can help diagnose Raynaud's disease.

Living with Raynaud's

People who are prone to Raynaud's can take measures to avoid some triggers.

The National Heart, Lung, and Blood Institute (NHLBI) suggest:

• wrapping up and keeping the house warm when temperatures are cold

• as far as possible, avoiding emotional stress

• exercise to promote a healthy lifestyle and reduce stress

• avoiding medicines and substances that trigger the symptoms

• limiting consumption of caffeine and alcohol

• not smoking

Homeopathy has good treatment for it. Getting medical help may prevent a worsening of symptoms and serious complications.

Causes

Exactly what causes Raynaud's remains unclear, but a hyperactivation of the sympathetic nervous system is known to cause an extreme narrowing of the blood vessels, known as vasoconstriction.

It can happen when the person enters a cold place, opens a freezer, or puts their hands in cold water.

Some people experience symptoms when faced with stress, even without an associated drop in temperature.

In healthy individuals, the circulatory system in the extremities, such as the fingers and toes, reacts to conserve heat in cold conditions. The small arteries that supply the skin with oxygen narrow to minimize the amount of heat lost through the exposed skin surface.

In people with Raynaud's disease, this narrowing is excessive. This is what causes the blood vessels almost to shut down.

Types

There are two types of Raynaud's disease: Primary and secondary.

Primary Raynaud's, or Raynaud's disease, is more common, and it affects people who do not have a secondary medical condition.

Secondary Raynaud's, or Raynaud's phenomenon or syndrome, results from an underlying medical issue. It is less common and tends to be more serious.

Causes of secondary Raynaud's

Causes of secondary Raynaud's include:

Diseases of the arteries: Atherosclerosis, a build-up of plaque in blood vessels, or Buerger's syndrome, a disorder where blood vessels in the hands and feet become inflamed, can cause Raynaud's symptoms. Primary pulmonary hypertension has also been linked to the disease.

Diseases of the connective tissue: Most people with scleroderma, a disease leading to hardened skin, have Raynaud's disease. The symptoms are commonly linked to lupus, rheumatoid arthritis, and Sjogren's syndrome, an autoimmune disease affecting glands.

Repetition or vibration: People whose hobbies or jobs require repetitive movements, such as typing or playing the guitar or piano, are at risk of developing Raynaud's symptoms. Those whose jobs involve using vibrating tools, such as a jackhammer, are also at risk.

Carpal tunnel syndrome: This condition puts pressure on the nerves traveling to the hand, and it increases susceptibility to Raynaud's symptoms.

Medications: Drugs that can induce Raynaud's include beta blockers, migraine medications containing ergotamine or sumatriptan, ADHD medications, some chemotherapy drugs, and some over-the-counter cold remedies.

Exposure to certain substances: Smoking narrows blood vessels and is a possible cause of Raynaud's. Other chemicals, such as vinyl chloride, may also play a role.

Injuries: Raynaud's can start after injuries such as frostbite, a broken wrist, or local surgery.

Raynaud's tends to affect females more than males. Primary Raynaud's normally starts between the ages of 15 years and 25 years, and secondary Raynaud's between 35 and 40 years.

The condition appears to run in families. A person a first-degree relative with Raynaud's is more likely to develop it.

Possible pathways of Raynaud's disease

The exact pathways of Raynaud's disease are not known, but it probably results from a combination of factors, and the combination of factors probably differs between individuals.

Other possibilities

Platelet activation appears to be higher in people with Raynaud's. Platelets are components in blood that clump together to help prevent bleeding.

Fibrinolysis is a natural reaction that takes place in the body to reduce clotting. Some studies have linked low levels of fibrinolysis with Raynaud's.

Oxidative stress is caused by an increase in free radicals, and it could be involved in Raynaud's.

Symptoms

Raynaud's disease causes blood vessels in the hands or feet to overreact to the cold, causing numbness and pain.

Raynaud's disease affects some people when they are exposed to the cold.

When temperatures drop, the blood vessels contract in the fingers or toes. This contraction causes hypoxia, or lack of oxygen, to the affected tissues. Fingers and toes will feel cold to the touch and possibly numb.

Often, the affected area will turn white, then later turn blue. Once the area is warmed and blood flow returns, a tingling sensation may accompany a red flush and, possibly, swelling. There may also be a painful, throbbing sensation.

Toes and fingers are most commonly affected, but Raynaud's can affect the nose, lips, and ears.

Some women may experience Raynaud's phenomenon of the nipples, especially during breastfeeding. It causes severe throbbing, similar to that experienced with a fungal Candida albicans infection, which can lead to misdiagnosis.

An episode normally lasts around 15 minutes, including the time it takes for the body to normalize.

Raynaud's feet

Raynaud's syndrome can affect either the hands or feet, or both.

To reduce the risk of an attack, it can help to keep the feet and hands warm, avoid smoking, and get enough exercise.

www.thehomeopathyclinic.co.in

If an attack starts, it may be reduced or prevented by warming the hands and feet at once, for example, by massaging them.

The feet and hands should be protected from cuts, bruises, and other injuries as far as possible, because the lack of circulation can make it harder for them to heal. Use lotion to prevent the skin from cracking, and make sure you have comfortable, well-fitting shoes.

Complications

Raynaud's is not normally life-threatening, but complications can occur.

Chilblains happen when there is a problem with the blood circulation, and Raynaud's is one possible cause. The skin becomes itchy, red, and swollen and it may feel hot, burning, and tender. Chilblains usually resolve in 1 to 2 weeks, but they can come back. Keeping the extremities warm can help prevent them. If the hands and feet become cold, warm them slowly, as too much heat can cause further damage.

If symptoms worsen and blood supply is substantially reduced for a long time, fingers and toes can become deformed.

If oxygen is completely cut off from the area, skin ulcers and gangrenous tissue can develop. Both of these complications are difficult to treat. They may eventually require amputation.

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globalstemcellcare · 2 years

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Stem Cell Therapy Information for Buerger's Disease

#globalstemcellcare #stemcelltreatmentinindia #stemcells #stemcelltherapy #treatmentthroughstemcell #treatmentforinternationalpatient #lowcoststemcelltreatment #MSCs #mesenchymalstemcell #Buerger #buergerdisease

#global stem cell care #MSCS #buerger disease #stem cell #stem cell treatment

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hcccardiologypa · 3 years

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Contact:

Address: 869 Main St, Darby, PA 19023

Phone: 610-461-2440

Website URL

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biogenericpublishers · 3 years

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Necrotizing Sialometaplasia of Palate by Radka Cholakova in Open Access Journal of Biogeneric Science and Research

Abstract

Necrotizing sialometaplasia (NS) is a rare, benign, inflammatory condition, occasionally with ulcers, which is self-limiting and affects mainly salivary tissue. Purpose: To present a case of NS associated with a systemic connective tissue disease in a female patient. Material/Methods: A 56-year-old female patient with ulcers of the palate, surrounded by a red halo, for 2 weeks, which did not resolve when treated with topical antiseptics. Clinical and radiological methods, together with pathohistological analysis, were used to make the diagnosis. Immunohistochemical analyses to diagnose patient's systemic disease were performed at a rheumatology clinic. Results: The definitive diagnosis was made based on a pathological examination and the tests performed in the rheumatology clinic, which found that this was a case of necrotizing sialometaplasia of the minor salivary glands and a concomitant systemic connective tissue disease. Conclusions: NS is a rare disease, with an excellent prognosis, without any possible preventive strategies.

Keywords: Necrotizing sialometaplasia; diseases of the minor salivary glands; tumour-like lesion

Introduction

Necrotizing Sialometaplasia (NS) is a rare, benign, inflammatory condition, sometimes with ulcers, which is self-limiting and affects mainly salivary tissue. This disease is classified as a “tumour-like lesion” in the WHO classification of salivary gland tumours. It was first described in 1973 by Abrams, Melrose and Howell [1], and in the following year, Dunlap and Barker reported five diagnosed cases [2]. This lesion can be confused with a malignant disease, thus resulting in unnecessary radical surgery.

Materials and Methods

A female patient of visible age of 56, corresponding to her calendar age, with complaints of a non-healing “aphthous ulcer”, which appeared 2 weeks earlier. Treatment with antiseptic mouthwashes and topical application of propolis was administered. The aphthous ulcer did not resolve, but the pain subsided over time. The initial examination showed two ulcers with dimensions of about 2 mm, located on either side of the median palatine suture, with a red halo around them. After treatment with SOLCOSERYL (MEDA Pharma GmbH & Co. KG, Germany), the ulcers resolved, but the red spots remained (Figure 1). The patient was referred for a CBCT in order to detect erosions in the palatine bone under the ulcers (Figure 2). The size of the formation did not decrease and, therefore, an excisional biopsy was performed in full thickness of one of the lesions, and then was provided for histopathological examination. The wound was covered with a PRF membrane and healed without complications. Pathological examination showed that there were cords of the lining multilayered squamous non-keratinizing epithelium, deep below it, without atypicality, and with rapid proliferation of granulation tissue, which covered the minor salivary gland ducts, with squamous cell metaplasia. The patient was a moderate smoker (up to 10 cigarettes/day). She had cholelithiasis without clinical manifestations. She had had surgery for a benign neoplasm of the breast several years before. Intermittent complaints of swelling and rash on the upper eyelids, erythematous rash on the nasal dorsum and photosensitivity were present, for which the patient had been admitted to a rheumatology clinic for examination. A systemic connective tissue disease was suspected there, as elevated total AHA, Anti-SS-A and Anti-SSB antibodies with very high titers and low C4 complement level, without significant proteinuria, were found. The dermatological examination showed pathological skin lesions of the nose with erythematous-edematous plaque, 6 cm in diameter. Sjögren's syndrome or systemic lupus erythematosus were suspected in this patient. The pathohistological examination of the minor salivary glands from the lower lip mucosa found that this was not a case of Sjögren's syndrome. Biopsy of the plaque of the nasal lesion was recommended.

Results

The definitive diagnosis was made based on a pathological examination and the tests performed in the rheumatology clinic, which found that this was a case of necrotizing sialometaplasia of the minor salivary glands and a concomitant systemic connective tissue disease.

Discussion

Necrotizing sialometaplasia is extremely rare, accounting for less than 1% of oral lesion biopsies [3]. The mean age of onset is 46 years, and the male:female ratio is 2.7:1 [4]. The disease is prevalent among the Caucasian race, with Caucasian:African American ratio of 5:1, according to Brannon [5]. In most cases (80%), the minor palatine salivary glands are affected. Although rarely, it can occur in the retromolar space, gingiva, lips, tongue, cheeks, and nasal cavity [1,6]. This disease can also affect the large salivary glands in more than 10% of the cases [3]. The etiopathogenesis of necrotizing sialometaplasia is unknown, but it is thought that the lesion develops as a result of previous ischemia in the salivary gland. In experimental models, disruption of the arterial blood supply to the salivary glands in rodents results in a NS-like histopathological picture. This disease is found in patients with sickle cell disease, Buerger's disease and Raynaud's phenomenon, which are all vasculopathies that predispose to ischemia. Other risk factors for the development of NS include: smoking (and alcohol consumption), use of cocaine and anabolic steroids, hot food, fellatio, traumatic vascular injury, and bulimia [7-9]. The synergistic action of NSAIDs and alcohol over a long period of time results in a change in the oral mucosa functions due to suppression of prostaglandin production and reduction in the blood supply to the minor salivary glands, which causes ischemic events [10]. The iatrogenic factors for the development of NS are the use of local infiltration anesthesia with an anesthetic with a higher concentration of correctives, intubation, bronchoscopy, local radiotherapy, as well as surgical procedures in the vicinity of the affected area [3,7,11,12], which, in the case described by us, had not been conducted for a period of more than 6 months. Senapati et al. [13] reported that NS is a manifestation of local vasculitis. It may be associated with other tumours, in particular Warthin's tumour, Abrisokov's tumour, lip cancer, rapidly growing malignant mesenchymal disease and salivary gland tumours. There is also a connection with previous upper respiratory tract diseases (chronic sinusitis and allergies) in the past few weeks. It is possible that ischemia is due to immune complexes, resembling the pathogenesis of erythema multiforme or benign trigeminal sensory neuropathy. In our case, levels of immune complexes in the body were elevated.

Anneroth and Hansen described the following five clinical stages in the development of necrotizing sialometaplasia: infarction, sequestration, ulceration, repair and healing [6]. A subacute variant of this condition was also described in the literature. Histological features are ischemic lobular necrosis of seromucinous glands, with maintenance of intact lobular architecture, despite coagulative necrosis of the mucinous acini. Pale acinar outlines often persist, but the cell nuclei are hypochromatic or absent. Mucin extravasation into the adjacent tissues triggers an inflammatory reaction dominated by histiocytes and granulation tissue. Within the necrotic lobules, the inflammatory component is often minimal, but is usually found in the surrounding tissues. Although squamous metaplasia of ducts and acini is typical (which makes the diagnosis challenging due to its similarity to malignancies), the metaplastic cells have benign nuclear morphology, with minimal pleomorphism or hyperchromatism and few mitotic figures. Nests of squamous epithelium, which usually have smooth contours, occasionally may have an irregular outline.

Pseudoepitheliomatous hyperplasia, where the overlying or adjacent epithelium is markedly hyperplastic, together with extensive ductal metaplasia, may resemble malignant condition of the epithelium, which could be the reason for misdiagnosis and radical ablative surgery. It may be difficult to distinguish NS from squamous cell carcinoma, low-grade mucoepidermoid carcinoma and oncocytic tumours. Specific histopathological characteristics may have some relation to the “age” of the lesion at biopsy. Coagulative necrosis is more common in “new” lesions, whereas fibrosis and squamous metaplasia are typical of “older” lesions. In our case, biopsy was taken nearly 2 weeks after the onset of the first symptoms, and therefore the changes correspond to an “old” lesion. Management of this disease includes monitoring, use of topical antiseptics, and pain control until recovery [3]. In the presence of predisposing factors, their correction is necessary.

Conclusion

This work was supported with grants from the URPC, University of Benin, Benin City.

More information regarding this Article visit: OAJBGSR

https://biogenericpublishers.com/pdf/JBGSR.MS.ID.00155.pdf https://biogenericpublishers.com/jbgsr.ms.id.00155.text/

For more open access journals click on https://biogenericpublishers.com/

#Necrotizing sialometaplasia #salivary glands #tumour #OAJBGSR #JBGSR #Radka Cholakova

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hccheart789 · 3 years

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HCC - Heart & Vascular Consultants

Address:

3379 Quakerbridge Rd Suite 202, Hamilton Township, NJ 08619

Phone:

609-393-0067

Website URL:

https://heartcareconsultants.com/

About US:

Heart & Vascular Care Consultants World-Class care, a heartbeat away. When it comes to cardiac care, seconds count. That’s why it’s good to know that wherever you find yourself, exceptional coronary and circulatory expertise is moments away at a nearby office of Heart & Vascular Care consultants. At Heart & Vascular Care Consultants, our practitioners treat every symptom, from the most commonplace to the most acute, with the same exacting diagnostic discipline. And we approach every patient and family with the same compassionate concern. It’s never been more important to have an experienced, trusted and responsive cardiologist nearby. Yet even a concern as common as spider veins, varicose veins, leg pain, leg swelling, leg discoloration, lymphedema can be the sign of a more critical underlying cardiac condition. Heart Catheterization. A now common and useful diagnostic procedure, heart catheterization allows your medical team to evaluate the condition of your heart. This is achieved by inserting a small tube into a blood vessel and injecting a dye that will allow them to see the flow through your arteries and identify any blockages. In most cases, you will be awake and resting comfortably throughout, complications are very rare and you will be home that evening. Preparation for this procedure is simple and begins 12 hours beforehand. Your Heart & Vascular Care doctor or nurse-practitioner will provide complete instructions beforehand.

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consultantsveinexpertsh · 3 years

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HCC - Cardiology Consultants & Vein Experts

Address:

40 Fuld St Suite 300, Trenton, NJ 08638

Phone:

(609) 695-4422

Website URL:

https://heartcareconsultants.com/

About US:

Whether your symptoms are superficial or serious, here you can be certain that your heart is in the right hands. At HCC - Cardiology Consultants & Vein Experts, our practitioners treat every symptom, from the most commonplace to the most acute, with the same exacting diagnostic discipline. And we approach every patient and family with the same compassionate concern.

Holter Monitor. This a live monitor of your heartbeat for evaluation of fast, slow, or irregular heartbeat. You will also indicate if you have any symptoms associated with the irregularity of your heartbeat.

Ambulatory Blood Pressure Monitor. This a live monitor of your blood pressure. The ambulatory blood pressure has been demonstrated to be more accurate than the office blood pressure and can tease out the anxious patient with elevated blood pressure at the office. This patient generated data will help us to tailor your hypertension therapy.

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veinstreatment0 · 3 years

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CONTACT

Address: 6122 Torresdale Ave, Philadelphia, PA 19135

Phone: 215-338-6677

Website URL:

https://heartcareconsultants.com/

About US

Our experienced cardiologists are prepared to care for patients of all ages and conditions. No matter what your condition – coronary artery disease, heart failure, heart valve disorders, rhythm disorders and even heart attack – we are committed to making sure that our patients feel better now and stay healthy in the future.

We understand that being diagnosed with a cardiac condition can be difficult, both physically and emotionally, and we strive to make your experience as easy and comfortable as possible. Our team will answer your questions at every step, fully explaining your options as we define the problem.

When HCC - Philadelphia Cardiology & Veins Treatment cardiologists diagnose a cardiac disorder, we will work closely with our patients and their family to determine the best treatment plan based on the patient’s age, condition and lifestyle. This treatment plan may include medical therapy, interventional cardiology, surgery, electrophysiology or a combination of these services.

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hyperbaricchamber4587-blog · 4 years

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Things To Know About Right Hyperbaric Chamber.

A hyperbaric chamber is designed to allow either one or more patients to be placed in an environment where pressure is more than the atmospheric pressure. This higher pressure subsequently leads to a higher concentration of oxygen which is associated with lots of therapeutic applications. A hyperbaric chamber boosts the amount of dissolved oxygen in the human tissues and blood, which includes the poorly vascularized parts.

The chamber can also be used to properly oxygenate damaged or diseased tissues as well as reducing the proliferation of specific bacteria which can only grow and thrive in oxygen-deficient environments. Some of the reputable therapeutic applications of the hyperbaric chamber include the treatment of decompression sickness, burns, skin injuries, carbon dioxide poisoning and post-radiotherapy treatment. Learn more about cryogenic chamber. Decompression sickness can usually occur when divers move up too quickly to the surface after diving deep into the water for a considerable amount of time without making the recommended decompression stops. The gas embolism can also affect personnel working in compressed air tanks, astronauts and airmen in high altitudes. The treatment by the hyperbaric chamber is highly effective in such cases.

A hyperbaric chamber is also used to treat burns and skin injuries which are proving difficult to heal as fast as expected. The hyperbaric oxygen is particularly helpful for severe burns. People who have gangrene, bedsores, Buerger’s disease and people with diabetes who are wounded can also benefit a lot from the use of hyperbaric chambers. The other use of the hyperbaric chamber is the post-radiotherapy treatment which refers to complications that usually after radiotherapy on patients with cancer like late radiation-induced tissue damage. The hyperbaric oxygen also improves the oxygen supply to tissues which could be damaged and subsequently prevent necrosis.

Patients who suffer from carbon II oxide poisoning can also benefit from the hyperbaric chamber. When people get exposed to carbon monoxide for some time, it can result in loss of consciousness. To get more details about hyperbaric chamber, click https://atlantahyperbariccenter.com/. This may later lead to memory problems, mood changes and personality disorders. The hyperbaric chamber is quite effective in reducing these effects. Hyperbaric chambers come in different configurations which include fixed, inflatable, casters and containerized.

The fixed hyperbaric chambers which are usually multipace are installed permanently in particular hospital units. The inflatable chambers, on the other hand, are quite lightweight, can be inflated anywhere and offer greater freedom of movement. The containerized chambers, on the other hand, are designed to be transported by specialized vehicles and trucks and can be installed on military vessels and drilling platforms. Learn more from https://en.wikipedia.org/wiki/Hyperbaric_medicine#Hyperbaric_chambers.

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cbdvapebattery-blog · 5 years

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Harmful Aftereffects of Cigarette Smoking

By smoking a cigarette, the nicotine within the cigarette, affects mental performance within 10 seconds, latter about it gradually affects one other elements of the body. From head to toe its effects are visible. In smoking, the carbon monoxide starts to dissolve in oxygen hence reducing its efficiency. The oxygen degree of the cells started initially to drop. The arteries become narrower and the blood pressure shoots up. A burning cigarette produces nearly four thousands chemical, of which 200 are extremely poisonous. One of them nearly 60 compounds are carcinogens in nature.

Pharmacological aftereffects of nicotine

· Cardiovascular system (C.V.S.): rise in B.P., tachycardia (accelerated heart rate), cutaneous vasoconstriction (skin turns paler because of less supply of blood due to narrowing of blood vessels)

· Ant diuretic: reduces urine volume due to A.D.H. (anti diuretic hormone) release

· Central nervous system (C.N.S.): stimulation, especially respiratory, vasomotor and emetic centres

· Adrenal: discharge adrenaline

· Automatic: transient stimulation, ultimately depression of most ganglia (nerve cells bodies)

Pharyngeal (throat) and bronchial irritation

· Bronchitis (inflammation of bronchi due to longterm contact with irritants)

· Pneumonia.

Peptic ulcer morality increased

Because of associated alcoholism incident of Cirrhosis increased

Carcinoma of following areas of the body increased

· Oesophagus

· Prostate

· Bronchus

· Bladder

Cardio-vascular disease exacerbated

· Myocardial ischemia (reduced blood supply to heart muscles)

· Buerger's disease (inflammation and clotting of veins and arteries of hands and feet)

Influence on fetus

· Smoking during pregnancy restricts growth and increases prenatal mortality rate

Individual peculiar symptoms

· Tobacco angina

· Atrial extrasystoles (premature electrical impulse in the heart)

· Hypoglycaemia (below normal blood glucose)

Current treatment for: smoking cessation, nicotine addiction, tobacco addiction or stop smoking programs are typical place. The success of these programs varies widely. Bupropion (AKA Zyban, Wellbutrin, Voxra, Budeprion or Aplenzin) is a common smoking cessation aid. Nicotine replacement nicotine blocking agents will also be frequently used. Good outcome statistics are hard to find. Recovery from nicotine addiction, cigarette smoking and tobacco addiction is difficult and on a par with heroin recovery!

There's also alternative medical treatments or therapies for recovery from nicotine or cigarettes addiction. There are herbs and supplements that help with smoking with 510 battery cessation and nicotine addiction. Individuals who are nicotine dependent or involved in smoking cessation can take advantage of these non-addicting, affordable herbal supplements. These herbal supplements are natural and can be used as needed. These therapies to take care of smoking or nicotine addiction, withdrawal, detox and/or cravings should be continued for at least 2 years. It takes that long to normalize the electrical and chemical changes in mental performance that occurred with ongoing nicotine addiction.

Behavioral and cognitive therapy is essential for any smoking cessation program. It would be wise. There are 12-Step programs for almost everything, including AA (Alcoholics Anonymous), NA (Narcotics Anonymous), CA (Cocaine Anonymous), OA (Overeaters Anonymous) and Nicotine Anonymous. Recovery from nicotine and smoking cessation are lifelong journeys.

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